The Synaptogenesys Suite



The Driving Principles

Acute stress impairs decision-making and self-control (Maier et al., 2015). Chronic stress damages neurons, shrinks the brain, and can lead to addiction (Ansell et al., 2012; Hathaway, 2012; Sinha, 2008). Traumatic stress inflicts permanent brain injury beyond acute and chronic stress (Nutt & Malizia, 2004; Bremner, 2006; Kroes et al., 2011).

“The brain is plastic, and there are ways to reverse some of the effects of stress,” (Park, 2012). “Learning new things prompts brain plasticity to restructure the brain and restore lost functionality” (Pearce, 2015). Synchronized light and sound stimulation may recover motor functions impaired by stroke and other traumatic brain injuries (Thaut et al., 2010; Trafton, 2014; da Silva et al., 2015; Reybrouk et al., 2018).

Extended dynamic learning stimulation (4 months) can significantly reverse brain harm from traumatic experiences, fostering resilience and improving life quality. New neurons generated daily in the adult brain, especially in the hippocampus, can be preserved through challenging learning (Shors, 2009). Pattern recognition-based learning enhances synaptogenesis, further supported by the structured Logic of the Synaptogenesys™ Suite (Mattson, 2014).

Engaging new neurons through learning boosts resilience and helps the brain rebound from stress (Annacker et al., 2016). UHL media dynamically facilitates this engagement.


The Suite

The Synaptogenesys Suite (Synaptogenesys) includes three therapeutic meditation modules designed to enhance brain function through synchronized, multimodal synaptogenesis. Each module is designed to stimulate more synapses than reside in the average, healthy adult human brain, and to promote cross-modal neurorehabilitation through coordinated, sequential engagement of neurotherapeutic activities.



The User's Experience

The suite’s modules feature ambient rain and flowing water sounds, with headphones boosting efficacy by approximately 1400% (Trafton, 2014). A binaural beat entrains the brain to theta for optimal learning, complemented by Mindfulness Meditation (Tang, Hölzel, & Posner, 2015). Pranayama yoga enhances awareness and neuroplasticity (Shors, 2009), while visualization techniques engage relevant synapses to improve performance (Trafton, 2014; Mattson, 2014).




Caveat:

Your brain will automatically apply a lot of the synaptic increase to strengthening existing neural pathways. On your behalf, as your brain so applies the increase, it also increases the magnetism of the Law of Attraction; which dictates that you actualize what you think-and-feel most frequently. Hence, it's very important to pay keen attention to things like your media diet, and your most consistent thought habits about yourself, and about other people. Remember the adage, "Garbage in; garbage out!", own your mind and heart, and use the instructions to your greatest advantage.



Demos  (Test Drive links open in new tabs/windows)
  Level I          
              Level II        Level III

These are the respective visual dynamics of each Synaptogenesys level.


Therapeutic Uses
Trauma Level:            
                              Life-impairing

                              Severe           
                      Extreme
______________________________________________________________________________
Cognitive Incline Potentials*

                                    Major
 		                               Mega                             Maxi   



Synaptogenesys Level One


 		   


Features 128 spheres and 64 quadrachords, inducing synchronized synaptogenesis and neuroplasticity. Ideal for mild brain impairments like Stroke, Concussion, or Chronic Fatigue Syndrome.

Duration: 30 minutes. 



Synaptogenesys Level Two

   

Expands to 128 spheres with 64 quadrachords, providing more intensive synaptogenesis and neuroplasticity. Suitable for severe impairments such as PTSD and TBI.

Duration: 30 minutes.


Synaptogenesys Level Three

   
Incorporates 360 spheres and 148 quadrachords with complex sonically-expressed Logic. Enhances structural plasticity and spatial awareness. Designed for extreme brain impairments like C-PTSD, TBI, and Hemispherectomy.

Duration: 58 minutes


Test drive any of the above demos whilst monitoring using EEG, and/or MEG to determine whether any level of Synaptogenesys may be effective for a client.













Manifold Benefits of Using Synaptogenesys

With recommended use, the below benefits may be experienced with Synaptogenesys, and all of our Ultra-High Learning solutions:
  • Dispersion of brain fog;
  • Increased capacity to concentrate, and to focus;
  • Improved short-term, and long-term memory;
  • Heightened general, spatial, and peripheral perception;
  • Overall increase in cognitive enhancement, and lucid thinking;
  • Increased physiological, (and, consequently), psychological resilience;
  • Increased stamina and endurance;
  • Mindfulness development; (and, as a result);
  • Increased inner peace and calm;
  • Increased vitality, and ability to manage stress in the moment;
  • Increased immune response, (favoring brain healing);
  • Increased general dopamine [chill out neurotransmitter] levels;
  • Decreased general cortisol [fight-or-flight hormone] levels;
  • Alleviation of a remarkable amount of the emotional gravity that accompanies Major Depressive Disorder;
  • Advances restoration of lost motor capacity consequent to stroke, and concussion;
  • (May advance) Alleviation of motor impairment from Stroke, as well as from Chronic Fatigue Syndrome and other autoimmune conditions affecting motor capacities.
                   

Post-Session Duration of Synaptogenesys-Elevated Functionality

Synaptogenesys boosts functionality for approximately 1.5 times the session duration (e.g., 45 minutes for Level I and II, 90 minutes for Level III). At least 8 sessions are recommended for the brain to begin to fully track [hence, to learn] the Sync Sequence.

Give your clients, (or yourself),
a  personal and competitive advantage;

the Synaptogenesys Advantage.
 




 






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References
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Bliss, T. V., Collingridge, G. L., & Morris, R. G. (2013). Synaptic plasticity in health and disease: introduction and overview. Philosophical transactions of the Royal Society of London. Series B, Biological Sciences, 369(1633), 20130129. https://doi.org/10.1098/rstb.2013.0129

Bremner, J. D. (2006). Traumatic stress: effects on the brain. Dialogues in Clinical Neuroscience, 8(4), 445–461.

Chen, Z., Haykin, S., Eggermont, J. J., Becker, S. (2007). Correlative Learning: A Basis for Brain and Adaptive Systems. John Wiley & Sons, Inc.

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Crum, K. I., Flanagan, J. C., Vaughan B, et al. (2021). Oxytocin, PTSD, and sexual abuse are associated with attention network intrinsic functional connectivity. Psychiatry Res. Neuroimaging.  Psychiatry Research Neuroimaging 316(11):111345. (Also available via) doi: 10.1016/j.pscychresns.2021.111345

Cunningham, R. (2019). Neuroplasticity: How the brain changes with learning. University of Queensland. https://solportal.ibe-unesco.org/articles/neuroplasticity-how-the-brain-changes-with-learning/

Estrada, B., Molina, N., Navarro, M. (2015). Neuropsychological and Physiological Outcomes Pre- and Post-EMDR Therapy for a Woman With PTSD: A Case Study. Journal of EMDR Practice and Research. 9. 174-187. 10.1891/1933-3196.9.4.174.

Graham, C. (2021). Brain connectivity is lower in adults with PTSD, or a history of sexual abuse. https://web.musc.edu/about/news-center/2021/10/27/van-dan-ptsd

Hanks, H. (2016). How To Overcome PTSD (Post Traumatic Stress Disorder).

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Hindley, N., Sanchez Avila, A., & Henstridge, C. (2023). Bringing synapses into focus: Recent advances in synaptic imaging and mass-spectrometry for studying synaptopathy. Frontiers in synaptic neuroscience, 15, 1130198. https://doi.org/10.3389/fnsyn.2023.1130198

Högberg, Göran. (2019). Post-Traumatic Stress Disorder: Neurobiology and Effects of Eye Movement Desensitization and Reprocessing.

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Kroes, M. C., Rugg, M. D., Whalley, M. G., & Brewin, C. R. (2011). Structural brain abnormalities common to post traumatic stress disorder and depression. Journal of Psychiatry & Neuroscience : JPN, 36(4), 256–265. https://doi.org/10.1503/jpn.100077

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Maier, S. U., Makwana, A. B., Hare, T. A. (2015). Acute Stress Impairs Self-Control in Goal-Directed Choice by Altering Multiple Functional Connections within the Brain’s Decision Circuits. Neuron 87, 621–631

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* Using any UHL product to avance personal cognitive enhancement also requires active, or focused learning. The instructions provide you with guidance in how to use focused learning to translate the above "Major", "Mega", and "Maxi" stimulation potentials of Levels I, II, and III, respectively, into corresponding levels of cognitive incline.










Revised July 18, 2024

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